Grants and Contracts Details
Description
Neuromuscular deficiencies during a jump-landing task are commonly studied in patients with chronic
ankle instability (CAI). Recent studies reported an altered sagittal-plane motion pattern in the ankle of
patients with CAI before landing, suggesting altered feed-forward neuromuscular control may be
associated with the development of CAI. However, there is no definitive explanation for the origin of the
feed-forward neuromuscular alteration. Emerging evidence suggests inhibition of the descending
corticospinal motor pathways in the tibialis anterior (TA) muscle may lead to altered feed-forward patterns
in CAI population. Determining these associations may illuminate why individuals with CAI display
altered neuromuscular control patterns during jump-landing tasks. Therefore, we aim to determine (1) if
differences exist in corticospinal excitability of the TA between CAI and healthy controls and (2)
associations between corticospinal excitability of the TA and sagittal-plane ankle motions exist during a
jump-landing task. The central hypothesis is that individuals with CAI will demonstrate a decrease in
corticospinal excitability of the TA compared to healthy controls, which will be associated with altered
feed-forward ankle motion patterns. The anticipated results of this study will help to explain
neurophysiological consequences of CAI and develop novel and effective rehabilitative approaches for
CAI.
Status | Finished |
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Effective start/end date | 8/18/15 → 8/17/16 |
Funding
- NATA Research & Education Foundation: $1,000.00
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