Amyloid β-peptide(1-42), oxidative stress, and alzheimer's disease

D. Allan Butterfield

doi:10.1007/978-1-84628-440-3_5

Amyloid β-peptide(1-42), oxidative stress, and alzheimer's disease

D. Allan Butterfield

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

2 Scopus citations

Abstract

Alzheimer's disease (AD) a progressive, age-related neurodegenerative disorder that affects memory, cognition, and speech, is present in more than 4 million persons. The number of cases of AD will significantly elevate, because the mean population in the United States is increasing [1]. AD is characterized pathologically by the presence of extracellular senile plaques, intracellular neurofibrillary tangles, and synapse loss. Senile plaques are composed of an amyloid beta-peptide (Aβ) core surrounded by dystrophic neurites.

Original language	English
Title of host publication	Abeta Peptide and Alzheimer's Disease
Subtitle of host publication	Celebrating a Century of Research
Pages	83-92
Number of pages	10
DOIs	https://doi.org/10.1007/978-1-84628-440-3_5
State	Published - 2007

ASJC Scopus subject areas

General Medicine

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10.1007/978-1-84628-440-3_5

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abstract = "Alzheimer's disease (AD) a progressive, age-related neurodegenerative disorder that affects memory, cognition, and speech, is present in more than 4 million persons. The number of cases of AD will significantly elevate, because the mean population in the United States is increasing [1]. AD is characterized pathologically by the presence of extracellular senile plaques, intracellular neurofibrillary tangles, and synapse loss. Senile plaques are composed of an amyloid beta-peptide (Aβ) core surrounded by dystrophic neurites.",

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AB - Alzheimer's disease (AD) a progressive, age-related neurodegenerative disorder that affects memory, cognition, and speech, is present in more than 4 million persons. The number of cases of AD will significantly elevate, because the mean population in the United States is increasing [1]. AD is characterized pathologically by the presence of extracellular senile plaques, intracellular neurofibrillary tangles, and synapse loss. Senile plaques are composed of an amyloid beta-peptide (Aβ) core surrounded by dystrophic neurites.

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Amyloid β-peptide(1-42), oxidative stress, and alzheimer's disease

Abstract

ASJC Scopus subject areas

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