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Chapter 25 Extracellular matrix adhesion: Biological, molecular, and pathogenic mechanisms

Research output: Contribution to journalArticlepeer-review

4 Scopus citations
Original languageEnglish
Pages (from-to)573-607
Number of pages35
JournalPrinciples of Medical Biology
Volume7
Issue numberC
DOIs
StatePublished - 1997

Bibliographical note

Funding Information:
molecules. This postulate is supported by the complexity of ECM molecules and integrin distributions in embryonic and adult tissues. Alternatively, different inte-grins may be linked transmembrane to different signaling pathways for the different physiological responses of cells on a complex matrix.

Funding

molecules. This postulate is supported by the complexity of ECM molecules and integrin distributions in embryonic and adult tissues. Alternatively, different inte-grins may be linked transmembrane to different signaling pathways for the different physiological responses of cells on a complex matrix.

FundersFunder number
Department of Neurosurgery
National Institutes of Health (NIH)T32-AG00105, CA27755, NS17139, AG02921, AG10213

    ASJC Scopus subject areas

    • General Biochemistry, Genetics and Molecular Biology

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