TY - JOUR
T1 - Delayed development of sensorineural hearing loss after neonatal hyperbilirubinemia
T2 - A case report with brain magnetic resonance imaging
AU - Worley, Gordon
AU - Erwin, Charles W.
AU - Goldstein, Ricki F.
AU - Provenzale, James M.
AU - Ware, Russell E.
PY - 1996
Y1 - 1996
N2 - Sensorineural hearing loss has long been known to be a clinical consequence of kernicterus. Brainstem auditory evoked potentials (BAEPs) that occur in hyperbilirubinemic infants, can be reversed in the neonatal pried by exchange transfusion. The case was reported in an infant with neonatal hyperbilirubinemia from hemolysis due to glucose-6-phosphate dehydrogenase (G6PD) deficiency and napthalene exposure. BAEPs showed that the baby had normal hearing at 30 decibels at 13 days of age, after exchange transfusions, but had developed profound bilateral sensorineural hearing loss by 7 months of age. The brain magnetic resonance imaging (MRI) findings at 7 months are also presented.
AB - Sensorineural hearing loss has long been known to be a clinical consequence of kernicterus. Brainstem auditory evoked potentials (BAEPs) that occur in hyperbilirubinemic infants, can be reversed in the neonatal pried by exchange transfusion. The case was reported in an infant with neonatal hyperbilirubinemia from hemolysis due to glucose-6-phosphate dehydrogenase (G6PD) deficiency and napthalene exposure. BAEPs showed that the baby had normal hearing at 30 decibels at 13 days of age, after exchange transfusions, but had developed profound bilateral sensorineural hearing loss by 7 months of age. The brain magnetic resonance imaging (MRI) findings at 7 months are also presented.
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U2 - 10.1111/j.1469-8749.1996.tb15090.x
DO - 10.1111/j.1469-8749.1996.tb15090.x
M3 - Article
C2 - 8631524
AN - SCOPUS:0029920897
SN - 0012-1622
VL - 38
SP - 271
EP - 277
JO - Developmental Medicine and Child Neurology
JF - Developmental Medicine and Child Neurology
IS - 3
ER -