Dietary vitamin E does not inhibit the promotion of liver carcinogenesis by polychlorinated biphenyls in rats

Howard P. Glauert, Zijing Lu, Amita Kumar, R. Petruta Bunaciu, Sandip Patel, Job C. Tharappel, Divinia N. Stemm, Hans Joachim Lehmler, Eun Y. Lee, Larry W. Robertson, Brett T. Spear

Research output: Contribution to journalArticlepeer-review

12 Scopus citations

Abstract

In this study, the effect of dietary vitamin E on the hepatic tumor-promoting activity of PCB-77 and PCB-153 in female Sprague-Dawley rats (175-200 g) was investigated. One week after diethylnitrosamine injection, rats were fed purified diets containing 10, 50, or 250 mg/kg vitamin E in the form of α-tocopheryl acetate. Starting 1 wk later, we injected rats i.p. with vehicle (corn oil) or PCB-77 or PCB-153 (300 μmol/kg) every 14 d for 4 injections. All rats were killed 10 d after the last PCB injection. The number and volume of placental glutathione S-transferase (PGST)-positive foci were increased by PCB-77 but not by PCB-153. Vitamin E did not affect the induction of PGST-positive foci. PCB-77, but not PCB-153, increased hepatic NF-κB activity. In conclusion, dietary vitamin E supplementation does not protect against the induction of altered hepatic focal lesions by PCBs.

Original languageEnglish
Pages (from-to)283-286
Number of pages4
JournalJournal of Nutrition
Volume135
Issue number2
DOIs
StatePublished - Feb 2005

Keywords

  • Altered hepatic foci
  • NF-κB
  • Polychlorinated biphenyls
  • Vitamin E

ASJC Scopus subject areas

  • Medicine (miscellaneous)
  • Nutrition and Dietetics

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