Do proteomics analyses provide insights into reduced oxidative stress in the brain of an Alzheimer disease transgenic mouse model with an M631L amyloid precursor protein substitution and thereby the importance of amyloid-beta-resident methionine 35 in Alzheimer disease pathogenesis?
Rukhsana Sultana, Renã A.S. Robinson, Miranda Bader Lange, Ada Fiorini, Veronica Galvan, Joanna Fombonne, Austin Baker, Olivia Gorostiza, Junli Zhang, Jian Cai, William M. Pierce, Dale E. Bredesen, D. Allan Butterfield
Research output: Contribution to journal › Article › peer-review
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