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Molecular mechanisms of Cr(VI)-induced carcinogenesis
Min Ding, Xianglin Shi
Toxicology and Cancer Biology
College of Medicine
Research output
:
Contribution to journal
›
Article
›
peer-review
116
Scopus citations
Overview
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Dive into the research topics of 'Molecular mechanisms of Cr(VI)-induced carcinogenesis'. Together they form a unique fingerprint.
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Medicine and Dentistry
Carcinogenesis
100%
Carcinogen
100%
Laboratory
50%
Gene Expression
50%
Cells
50%
Mechanism of Action
50%
DNA Damage
50%
Transcription Factor
50%
Hypoxia Inducible Factor 1
50%
Whole Animal
50%
Protein P53
50%
Vasculotropin
50%
Programmed Cell Death
50%
Cell Growth
50%
Ascorbic Acid
50%
Protein Phosphorylation
50%
Alternative Complement Pathway C3 C5 Convertase
50%
Transcription Factor AP 1
50%
Cell Organelle
50%
Flavoprotein
50%
Biochemistry, Genetics and Molecular Biology
Carcinogenesis
100%
Reduction (Chemistry)
100%
Animal
50%
Alpha Oxidation
50%
Gene Expression
50%
DNA Damage
50%
Transcription Factor
50%
Molecule
50%
Apoptosis
50%
Hypoxia Inducible Factor 1
50%
P53
50%
Cell Growth
50%
Tyrosine Phosphorylation
50%
Vascular Endothelial Growth Factor
50%
Vitamin C
50%
Radical Reaction
50%
AP-1 Transcription Factor
50%
Flavoenzyme
50%
Pharmacology, Toxicology and Pharmaceutical Science
Carcinogenesis
100%
Free Radical
100%
Hypoxia Inducible Factor 1
50%
Vasculotropin
50%
Protein P53
50%
Growth Disorder
50%
Ascorbic Acid
50%
Transcription Factor
50%
Transcription Factor AP 1
50%
Alternative Complement Pathway C3 C5 Convertase
50%
Flavoprotein
50%