Neutrophil lysosomal degradation of human CRP: CRP-derived peptides modulate neutrophil function

E. G. Shephard, R. Anderson, S. M. Beer, C. E. Jansen Van Rensburg, F. C. De Beer

Research output: Contribution to journalArticlepeer-review

32 Scopus citations

Abstract

Hydrolysis of human C-reactive protein (CRP) at pH 4.5 and pH 7.4 with neutrophil-derived lysosomal enzymes yielded 10% trichloroacetic acid soluble peptides (M(r) < 14,000). These peptides inhibited neutrophil superoxide production, chemotaxis, degranulation and phagocytosis at 2 μg/ml. This inhibition was not observed with native CRP or intermediate peptides (M(r) > 14,000). CRP peptides (M(r) < 14,000) also caused a dose-related inhibition of Quin-2 fluorescence indicating interference with intracellular calcium movements during cell activation. These results point to a potential regulatory role for CRP-derived degradation products on neutrophils during inflammation.

Original languageEnglish
Pages (from-to)139-145
Number of pages7
JournalClinical and Experimental Immunology
Volume73
Issue number1
StatePublished - 1988

ASJC Scopus subject areas

  • Immunology and Allergy
  • Immunology

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