Abstract
Intrauterine growth restriction (IUGR) leads to reduced birth weight and the development of metabolic diseases such as Type 2 diabetes in adulthood. Mitochondria dysfunction and oxidative stress are commonly found in key tissues (pancreatic islets, liver, and skeletal muscle) of IUGR individuals. In this review, we explore the role of oxidative stress in IUGR-associated diabetes etiology.
| Original language | English |
|---|---|
| Pages (from-to) | 348-359 |
| Number of pages | 12 |
| Journal | Physiology |
| Volume | 33 |
| Issue number | 5 |
| DOIs | |
| State | Published - 2018 |
Bibliographical note
Publisher Copyright:© American Physiological Society. All rights reserved.
Funding
This work was supported by National Institute of Diabetes and Digestive and Kidney Diseases Grant R01 DK-114054.
| Funders | Funder number |
|---|---|
| National Institute of Diabetes and Digestive and Kidney Diseases | R01DK114054 |
UN SDGs
This output contributes to the following UN Sustainable Development Goals (SDGs)
-
SDG 3 Good Health and Well-being
ASJC Scopus subject areas
- Physiology
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